Assessment of α-Synuclein Secretion in Mouse and Human Brain Parenchyma

نویسندگان

  • Evangelia Emmanouilidou
  • Dimitris Elenis
  • Themis Papasilekas
  • Georgios Stranjalis
  • Kyriaki Gerozissis
  • Penelopi C. Ioannou
  • Kostas Vekrellis
چکیده

Genetic, biochemical, and animal model studies strongly suggest a central role for α-synuclein in the pathogenesis of Parkinson's disease. α-synuclein lacks a signal peptide sequence and has thus been considered a cytosolic protein. Recent data has suggested that the protein may be released from cells via a non-classical secretory pathway and may therefore exert paracrine effects in the extracellular environment. However, proof that α-synuclein is actually secreted into the brain extracellular space in vivo has not been obtained. We developed a novel highly sensitive ELISA in conjugation with an in vivo microdialysis technique to measure α-synuclein in brain interstitial fluid. We show for the first time that α-synuclein is readily detected in the interstitial fluid of both α-synuclein transgenic mice and human patients with traumatic brain injury. Our data suggest that α-synuclein is physiologically secreted by neurons in vivo. This interstitial fluid pool of the protein may have a role in the propagation of synuclein pathology and progression of Parkinson's disease.

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عنوان ژورنال:

دوره 6  شماره 

صفحات  -

تاریخ انتشار 2011